Session III: Invasive Diagnosis and Treatment-6155-Workshop #4- SVT and VT Invasive-Noninvasive Correlation

Video Transcription

Video Summary

Case 1: The best interpretation of the results is that adenosine is making the conduction through the accessory pathway better. The accessory pathway conduction improves due to the hyperpolarizing effects of adenosine. This suggests that the ablation was not yet successful.<br /><br />Case 2: The tachycardia is most likely caused by non-reentrant junctional tachycardia due to isoproterenol. The administration of isoproterenol facilitates the automaticity of the junctional tissue and triggers the tachycardia.<br /><br />Case 3: The best target for ablation is the site of earliest ventricular activation during tachycardia, which suggests that the tachycardia is arising from that site.<br /><br />Case 4: The QRS complex widens because adenosine causes accelerated automaticity in the damaged pathway. Adenosine can enhance automaticity in damaged or ischemic myocardium, leading to widening of the QRS complex.<br /><br />Case 5: The ablation has caused complete AV block. There is no retrograde accessory pathway conduction seen during pacing, indicating that the ablation was successful in eliminating the accessory pathway.

Keywords

adenosine

conduction

accessory pathway

ablation

tachycardia

isoproterenol

automaticity

junctional tissue

target

QRS complex

AV block